Author

Milka Koupenova

University of Massachusetts Chan Medical SchoolORCID 0000-0001-5934-8990Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Genomics· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Approach:experimental assay· 2 Assay:ATAC-seq· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

Amada D. CaliziD· 1 shared Anastassiia VertiiiD· 1 shared Ann Marshak‐RothsteiniD· 1 shared Carolina Salomão LopesiD· 1 shared Haibo LiuiD· 1 shared Heather LearnardiD· 1 shared Jane ChupriniD· 1 shared John F. KeaneyiD· 1 shared

Activity by Year

2023-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperGenomics

Amada D. CalizORCID

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2 papers

Recent·Most cited

ImmunologyJournal of Investigative DermatologyPeer-reviewedMar 28, 2026

Endothelial Cell Expression of STINGV154M Gain-of-Function Mutation Delays the Resolution of UVB-induced Skin Injury

Jane Chuprin, Carolina Salomão Lopes, Kevin MingJie Gao…·Mehdi Rashighi

This study shows that endothelial cells with a STINGV154M mutation cause persistent skin inflammation after UVB exposure, linking STING activation to impaired myeloid cell recruitment and delayed inflammation resolution.

DOI 10.1016/j.jid.2026.03.014

mousebulk RNA-seq

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GenomicsbioRxivPreprintOct 11, 2023

Inflammatory stress-mediated chromatin changes underlie dysfunction in endothelial cells.

Haibo Liu, Amada D. Caliz, Heather Learnard…·Anastassiia Vertii

The paper investigates chromatin changes in human endothelial cells under inflammatory stress using ATAC-seq and RNA-seq, finding correlations between chromatin accessibility and gene expression related to endothelial dysfunction.

1 citation

humanATAC-seq

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