Author

Huanjie Sheng

University of California, BerkeleyORCID 0000-0001-6047-0053Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Genetics· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Organism:mouse· 1 Approach:statistical model· 1

Frequent Co-Authors

Peter H. SudmantiD· 2 shared A.R. Friedman· 1 shared Adiel Charbash· 1 shared Albert J. BeckeriD· 1 shared Alon FriedmaniD· 1 shared Andrew G. DilliniD· 1 shared Ariana Neelufar MoghbeliD· 1 shared Barry P. Hart· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersGenetics

Peter H. SudmantORCID

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2 papers

Recent·Most cited

GeneticsNature CommunicationsPeer-reviewedOct 3, 2022

Tissue-specific impacts of aging and genetics on gene expression patterns in humans

Ryo Yamamoto, Ryan Chung, Juan Manuel Vázquez…·Peter H. Sudmant

This study quantifies how aging and genetics influence gene expression across 27 human tissues, finding tissue-specific differences in their contributions and identifying signatures of aging and selection.

79 citationstop 1%

humanbulk RNA-seq

𝕏 PostBluesky

NeurosciencebioRxivPreprintFeb 1, 2019

Blood-brain barrier dysfunction in aging induces hyper-activation of TGF-beta signaling and chronic yet reversible neural dysfunction

Vladimir V. Senatorov, A.R. Friedman, Dan Z. Milikovsky…·Daniela Kaufer

This study shows BBB dysfunction in aging leads to TGF-beta hyperactivation in astrocytes, causing neural dysfunction, which can be reversed by inhibiting TGF-beta signaling.

13 citations

humanmouse

𝕏 PostBluesky