Author

Rahul Kumar

DR. B.R.A. Institute Rotary Cancer HospitalORCID 0000-0001-6585-6780Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Rahul Kumar4 papers · National Institutes of Health

Biology Disciplines

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Bioinformatics· 1 Genomics· 1

Signature Assays, Organisms & Methods

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Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Assay:CRISPR screen· 1 Research Focus:database / resource· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Research Focus:drug response / drug discovery· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Ambuj KumariD· 1 shared Avik SenguptaiD· 1 shared Harsh GoeliD· 1 shared Kavita KundaliD· 1 shared Keerthana Vinod Kumar· 1 shared Mayilaadumveettil NishanaiD· 1 shared Pranay TanwariD· 1 shared R Kunjulakshmi· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

9 top collaborators · 18 mutual co-author links

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1 shared paperGenomics

Ambuj KumarORCID

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2 papers

Recent·Most cited

GenomicsbioRxivPreprintMay 19, 2023

AMLdb: A comprehensive multi-omics platform to understand the pathogenesis and discover biomarkers for acute myeloid leukemia

Keerthana Vinod Kumar, Ambuj Kumar, Kavita Kundal…·Rahul Kumar

AMLdb is a multi-omics platform for AML research that identifies potential therapeutic targets, including five genes, with ENO1 as a novel candidate.

1 citation

humanbulk RNA-seq

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BioinformaticsbioRxivPreprintApr 23, 2021

Computational investigation reveals that the mutant strains of SARS-CoV2 are highly infectious than wildtype

Rakesh Kumar, Rahul Kumar, Harsh Goel, Pranay Tanwar

This study uses molecular dynamics simulations to show that SARS-CoV2 mutant strains have higher binding affinity and stability than the wildtype, contributing to their increased infectivity.

DOI 10.1101/2021.04.23.441125

protein structurestatistical model

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