Author

Congru Li

Karolinska InstitutetORCID 0000-0001-7023-5962Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:scRNA-seq· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:development & morphogenesis· 1

Frequent Co-Authors

Elisabet Stener‐VictoriniD· 3 shared Qiaolin DengiD· 3 shared Claes OhlssoniD· 2 shared Emilie DerisoudiD· 2 shared Eva Lindgren· 2 shared Gustaw ErikssoniD· 2 shared Haojiang LuiD· 2 shared Hong JiangiD· 2 shared

Activity by Year

2024-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 40 mutual co-author links

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3 shared papersPhysiology

Elisabet Stener‐VictorinORCID

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3 papers

Recent·Most cited

PhysiologybioRxivPreprintMar 17, 2026

Cell type-resolved transcriptomic map of skeletal muscle in women with polycystic ovary syndrome

Tina Gorsek Sparovec, Gustaw Eriksson, Congru Li…·Elisabet Stener‐Victorin

This study creates a single-nuclei transcriptomic atlas of skeletal muscle in women with PCOS, identifying cell-type-specific dysregulation and therapeutic targets.

DOI 10.64898/2026.03.13.711523

humanscRNA-seq

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Developmental BiologyAdvanced SciencePeer-reviewedJan 20, 2026

Decoding human placental cellular and molecular responses to maternal obesity and fetal growth

Hong Jiang, Emilie Derisoud, Denise Parreira…·Qiaolin Deng

The paper identifies placental cell-type-specific transcriptional responses to maternal obesity and fetal growth using single-nucleus RNAseq and microfluidic organoids.

2 citationstop 2%

humanscRNA-seq

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PhysiologyAdvanced SciencePeer-reviewedJul 29, 2024

Dissecting the Impact of Maternal Androgen Exposure on Offspring Health through Targeting the Androgen Receptor in Developmental Programming

Haojiang Lu, Hong Jiang, Congru Li…·Qiaolin Deng

This study investigates how maternal androgen exposure affects placental development in a PCOS-like mouse model and identifies trophoblast dysfunction as a key mechanism.

25 citationstop 1%

mouseDNA methylation

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