Author

Bengt Westermark

Uppsala UniversityORCID 0000-0001-7153-5545Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1

Frequent Co-Authors

Anna SegermaniD· 1 shared Carl‐Henrik HeldiniD· 1 shared Erika DalmoiD· 1 shared Kohei MiyazonoiD· 1 shared Mia NiklassoniD· 1 shared Ryo Tanabe· 1 shared Verónica RendoiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

7 top collaborators · 9 mutual co-author links

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1 shared paperCancer Biology

Anna SegermanORCID

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2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintApr 7, 2026

Inhibition of HVEM suppresses growth and invasion of mesenchymal glioblastoma

Ryo Tanabe, Bengt Westermark, Carl‐Henrik Heldin, Kohei Miyazono

This study identifies HVEM as a driver of mesenchymal glioblastoma and shows that an anti-HVEM nanobody inhibits tumor growth and invasion.

DOI 10.64898/2026.01.16.699674

humanbulk RNA-seq

Cancer BiologyInternational Journal of Molecular SciencesPeer-reviewedApr 23, 2025

BMP4 induces a p21-dependent cell state shift in glioblastoma linking mesenchymal transition to senescence

Mia Niklasson, Erika Dalmo, Anna Segerman…·Bengt Westermark

This study shows BMP4 induces p21-dependent senescence in glioblastoma cells, especially in therapy-resistant mesenchymal-like clones, linking mesenchymal transition to senescence.

10 citations

humanbulk RNA-seq