Author

Michael J. Hurley

Aligning Science Across Parkinson'sORCID 0000-0001-8022-9283Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Approach:statistical model· 1

Frequent Co-Authors

Ali RoghanianiD· 1 shared Anthony H.V. SchapiraiD· 1 shared Björn FrendéusiD· 1 shared Elisa MenozziiD· 1 shared Hervé M. BlottièreiD· 1 shared James M. Hennegan· 1 shared Jane MacnaughtaniD· 1 shared Jessica L. TeelingiD· 1 shared

Activity by Year

2023-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperNeuroscience

Ali RoghanianORCID

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2 papers

Recent·Most cited

NeurosciencebioRxivPreprintNov 18, 2025

Deletion or Targeted Blockade of FcγRIIb (CD32b) Impairs α-Syn Propagation In-Vivo

James M. Hennegan, Michael J. Hurley, Kerry L. Cox…·Jessica L. Teeling

This study shows that FcγRIIb promotes α-syn propagation and neurotoxicity in vivo, suggesting it as a therapeutic target for synucleinopathies.

DOI 10.1101/2025.11.18.687143

mouseexperimental assay

NeuroscienceBrain CommunicationsPeer-reviewedJan 1, 2023

α-synuclein expression in response to bacterial ligands and metabolites in gut enteroendocrine cells

Michael J. Hurley, Elisa Menozzi, Sofia Koletsi…·Anthony H.V. Schapira

This study shows bacterial ligands and metabolites increase α-synuclein levels in gut enteroendocrine cells, potentially contributing to Parkinson's disease pathology.

22 citations

mousebulk RNA-seq