Author

Judith Favier

InsermORCID 0000-0001-8190-5853Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Judith Favier1 paper

Biology Disciplines

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Cancer Biology· 2 Biochemistry· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Research Focus:differential expression· 2 Research Focus:gene regulatory network inference· 2 Organism:mouse· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:drug response / drug discovery· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

Louis James Maher IIIiD· 2 shared Alexander R. OpotowskyiD· 1 shared Alexandre BuffetiD· 1 shared Amelia OleagaiD· 1 shared Ana Belén Moreno-CárdenasiD· 1 shared Ana González GarcíaiD· 1 shared Anand VaidyaiD· 1 shared Ángel M. Martínez‐MontesiD· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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2 shared papersBiochemistry

Louis James Maher IIIORCID

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3 papers

Recent·Most cited

BiochemistryiSciencePeer-reviewedMay 1, 2026

Unbiased CRISPR Synthetic Lethal Screening for Genetic Vulnerabilities in Succinate Dehydrogenase (SDH)-loss Model of Paraganglioma

Fatimah J. Al Khazal, Michael J. Emch, Cristina Correia…·Louis James Maher III

The paper uses CRISPR screening to identify genetic vulnerabilities in SDH-deficient paraganglioma cells, finding the neddylation pathway as a potential therapeutic target.

DOI 10.1016/j.isci.2026.115885

mousegenetic perturbation / CRISPR

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Cancer BiologybioRxivPreprintAug 1, 2025

Distinct proteomic and acylproteomic adaptations to succinate dehydrogenase loss in two cell contexts

Sherry X. Zhou, Benjamin J. Madden, Cristine M. Charlesworth…·Louis James Maher III

This study compares proteomic and acylproteomic changes in two cell types after SDH loss, finding distinct adaptations that may explain tissue-specific tumorigenesis.

1 citation

mouseproteomics

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Cancer BiologyCancer DiscoveryPeer-reviewedApr 8, 2025

Convergent genetic adaptation in human tumors developed under systemic hypoxia and in populations living at high altitudes

Carlota Arenillas, Lucía Celada, José Ruiz-Cantador…·Rodrigo Almeida Toledo

This study links EPAS1HIF2 genetic adaptations in high-altitude populations to similar mutations in hypoxia-driven tumors, showing shared mechanisms in hypoxia response and tumorigenesis.

14 citations

humanbulk RNA-seq

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