Patricio Opazo

The paper shows that deleting newly formed dendritic spines after learning disrupts memory formation, but not existing memories or new learning.

The paper shows that neurons in Alzheimer's disease models can compensate for dendritic spine loss through structural plasticity, including spine enlargement and regeneration.

This study shows that constitutive ghrelin receptor activity regulates AMPA receptor trafficking and synaptic plasticity, and its inhibition impairs memory.