Author

Julie A. Klaric

Brandeis UniversityORCID 0000-0001-8279-2467Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:DNA methylation· 2 Approach:experimental assay· 2 Assay:ChIP-seq· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1 Organism:E. coli· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1

Frequent Co-Authors

Arianna D. Reuven· 1 shared Carmine Ceraolo· 1 shared David J. GlassiD· 1 shared Eli PerriD· 1 shared Estelle VincendeauiD· 1 shared Leonie von Ondarza· 1 shared Mason J. Towne· 1 shared Nikhil VenkateshiD· 1 shared

Activity by Year

2021-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperGenetics

Arianna D. Reuven

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2 papers

Recent·Most cited

Exclusion from DNA double-strand breaks is a pervasive feature of the hnRNP family

Estelle Vincendeau, Leonie von Ondarza, Julie Klaric, Nikhil Venkatesh, Carmine Ceraolo, Stephanie Panier

The paper shows that most hnRNP proteins are excluded from DNA double-strand breaks, with hnRNPH1's exclusion dependent on its GYR domain and independent of recruitment pathways.

bioRxiv · Nov 3, 2026 · PreprintCell Biology

DNA damage-signaling, homologous recombination and genetic mutation induced by 5-azacytidine and DNA-protein crosslinks in Escherichia coli

Julie A. Klaric, David J. Glass, Eli Perr, Arianna D. Reuven, Mason J. Towne, Susan T. Lovett

This study investigates how 5-azacytidine induces DNA damage, homologous recombination, and genetic mutations in E. coli, revealing Dcm-dependent and -independent mechanisms of mutagenesis and DNA repair.

Mutation research. Fundamental and molecular mechanisms of mutagenesis · Jan 1, 2021Peer-reviewedGenetics