Author

Stephanie Panier

Max Planck Institute for Biology of Ageing, Cologne, GermanyORCID 0000-0002-7941-2846Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Cell Biology· 2

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:chromatin & epigenetic regulation· 2 Organism:human· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Assay:ChIP-seq· 1 Assay:DNA methylation· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

Anna Eva Koch· 1 shared Carmine Ceraolo· 1 shared Estelle VincendeauiD· 1 shared Francesca Pandolfo· 1 shared Jessica Spindler· 1 shared Johanna I. BihleriD· 1 shared Joshua J. CooniD· 1 shared Julie KlariciD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCell Biology

Anna Eva Koch

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Exclusion from DNA double-strand breaks is a pervasive feature of the hnRNP family

Estelle Vincendeau, Leonie von Ondarza, Julie Klaric, Nikhil Venkatesh, Carmine Ceraolo, Stephanie Panier

The paper shows that most hnRNP proteins are excluded from DNA double-strand breaks, with hnRNPH1's exclusion dependent on its GYR domain and independent of recruitment pathways.

bioRxiv · Nov 3, 2026 · PreprintCell Biology

SLX4IP acts in parallel to FANCM to limit BLM-dependent replication stress at ALT telomeres

Jessica Spindler, Francesca Pandolfo, Anna Eva Koch, Priscilla Piccirillo, Johanna I. Bihler, Marcel Morgenstern, Sandra Buschbaum, Joshua J. Coon, Robert Hänsel‐Hertsch, Kavi P.M. Mehta, Stephanie Panier

This study identifies SLX4IP as a suppressor of BLM-driven replication stress at ALT telomeres and reveals its parallel function with FANCM to regulate telomere maintenance.

bioRxiv · May 28, 2025 · PreprintCell Biology