Author

Cecilia Poderoso

Consejo Nacional de Investigaciones Científicas y TécnicasORCID 0000-0001-8606-2840Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 2 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:human· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

Paula Mariana MalobertiiD· 2 shared Yanina BenzoiD· 2 shared Celina MoralesiD· 1 shared Clarisa MarotteiD· 1 shared Denisse Ferrer ViñalsiD· 1 shared Fabrizio G. Arona· 1 shared Gabriela Alicia BergiD· 1 shared Georgina Blanco· 1 shared

Activity by Year

2021-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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2 shared papersCell Biology

Paula Mariana MalobertiORCID

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3 papers

Recent·Most cited

Cell BiologyJournal of the Endocrine SocietyPeer-reviewedSep 3, 2026

Angiotensin II and cAMP signaling pathways regulate mitochondrial biogenesis and activity in human adrenocortical cells.

Matías A. Belluno, Fabrizio G. Arona, Katia E. Helfenberger…·Cecilia Poderoso

This study shows that Ang II and cAMP signaling enhance mitochondrial biogenesis and activity in human adrenocortical cells, improving bioenergetic capacity for steroidogenesis.

DOI 10.1210/jendso/bvag218

humanstatistical model

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Cell BiologybioRxivPreprintAug 10, 2026

Unraveling a fine balance between ferroptosis, lipid metabolism, and hormonal protection in Leydig cell steroidogenesis

Yanina Benzo, Melina A. Dattilo, Maria A Raggio…·Paula Mariana Maloberti

This study investigates ferroptosis in Leydig cells, showing it disrupts steroidogenesis and reveals hormone-mediated protection mechanisms.

DOI 10.64898/2026.07.03.736405

humanbulk RNA-seq

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Molecular BiologyPharmacological ResearchPeer-reviewedDec 9, 2021

Buenos Aires, June 2021Inhibition of Mitochondrial Fission by Drp-1 Blockade by Short-Term Leptin and Mdvi-1 Treatment Improves White Adipose Tissue Abnormalities in Obesity and Diabetes

Paola Vanesa Finocchietto, H. Perez, Georgina Blanco…·M.C. Carreras

This study shows that inhibiting Drp-1 fission with leptin and mdivi-1 improves mitochondrial function and adipose tissue abnormalities in obese and diabetic mice.

42 citationstop 4%

mouseproteomics

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