Author

Rachael A. Vaubel

Mayo ClinicORCID 0000-0001-8842-5975Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:gene regulatory network inference· 2 Organism:human· 2 Assay:proteomics· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1

Frequent Co-Authors

Danielle M. BurgenskeiD· 2 shared Forest M. WhiteiD· 2 shared Jann Nagina SarkariaiD· 2 shared Terry C. BurnsiD· 2 shared Alicia D. D’SouzaiD· 1 shared Allison K. LowmaniD· 1 shared Ana Valeria CastroiD· 1 shared Andrew Edward SloaniD· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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2 shared papersSystems Biology

Danielle M. BurgenskeORCID

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3 papers

Recent·Most cited

Systems BiologyNature CommunicationsPeer-reviewedAug 8, 2026

Uncovering the signaling networks of disseminated glioblastoma cells in vivo with INSIGHT

Ryuhjin Ahn, Alicia D. D’Souza, Lawrence Long…·Forest M. White

The paper introduces INSIGHT, a platform for phosphoproteomic analysis of rare cell subpopulations, and applies it to study signaling changes in disseminated glioblastoma cells in vivo.

1 citation

mouseproteomics

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Cancer BiologybioRxivPreprintNov 10, 2025

Systems analysis uncovers early temozolomide responses and peptide antigens in glioblastoma

Elizabeth Y. Choe, Owen K. Leddy, Cécile Riviere-Cazaux…·Forest M. White

This study uses a multi-omics approach to analyze early TMZ responses in GBM cells, identifying TMZ-associated peptide antigens and immune-related changes.

DOI 10.1101/2025.11.10.687735

humanbulk RNA-seq

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Cancer BiologybioRxivPreprintJul 11, 2025

Tumor-initiating genetics and therapy drive divergent molecular evolution in IDH-mutant gliomas

Tamrin Chowdhury, C. Mircea S. Tesileanu, Emre Kocakavuk…·Roel G.W. Verhaak

This study identifies genetic changes in IDH-mutant gliomas that correlate with treatment response and tumor evolution using longitudinal genomic data from 206 patient samples.

6 citations

humanwhole-genome sequencing

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