Author

Nazgol‐Sadat Haddadi

University of Massachusetts Chan Medical SchoolORCID 0000-0001-8854-8182Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Immunology· 3

Signature Assays, Organisms & Methods

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Research Focus:cell-cell communication· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:spatial transcriptomics· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Approach:deep learning· 1 Research Focus:differential expression· 1

Frequent Co-Authors

Khashayar AfshariiD· 3 shared Mehdi RashighiiD· 3 shared John E. HarrisiD· 2 shared Manuel G. GarberiD· 2 shared Mridushi DagaiD· 2 shared Andressa Lumi AkabaneiD· 1 shared Ann Marshak‐RothsteiniD· 1 shared April DengiD· 1 shared

Activity by Year

2026-2026 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 32 mutual co-author links

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3 shared papersImmunology

Khashayar AfshariORCID

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3 papers

Recent·Most cited

ImmunologyScientific ReportsPeer-reviewedJul 22, 2026

Spatial characterization of interface dermatitis in cutaneous lupus reveals novel chemokine ligand-receptor pairs that drive disease

Saeed Shakiba, Nazgol‐Sadat Haddadi, Khashayar Afshari…·Jillian M. Richmond

This study identifies novel chemokine ligand-receptor pairs involved in immune cell recruitment in cutaneous lupus by combining spatial transcriptomics, proteomics, and immunological assays.

7 citationstop 1%

humanspatial transcriptomics

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ImmunologyNature ImmunologyPeer-reviewedApr 24, 2026

A Spatially Coordinated Keratinocyte-Fibroblast Circuit Recruits MMP9+ Myeloid Cells to Drive IFN-I-Driven Inflammation in Photosensitive Autoimmunity

Yuqing Wang, Khashayar Afshari, Nazgol‐Sadat Haddadi…·Manuel G. Garber

This study identifies MMP9+ CD14+ myeloid cells as key drivers of UVB-induced IFN-I-driven inflammation in cutaneous lupus and dermatomyositis through a keratinocyte-fibroblast-myeloid circuit.

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humanscRNA-seq

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ImmunologyJournal of Investigative DermatologyPeer-reviewedMar 28, 2026

Endothelial Cell Expression of STINGV154M Gain-of-Function Mutation Delays the Resolution of UVB-induced Skin Injury

Jane Chuprin, Carolina Salomão Lopes, Kevin MingJie Gao…·Mehdi Rashighi

This study shows that endothelial cells with a STINGV154M mutation cause persistent skin inflammation after UVB exposure, linking STING activation to impaired myeloid cell recruitment and delayed inflammation resolution.

DOI 10.1016/j.jid.2026.03.014

mousebulk RNA-seq

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