Olivier Rohr

This study identifies KLF16 as a repressive transcription factor that inhibits HIV-1 transcription by competing with Sp1 and recruiting repressive complexes.

This study identifies lysine K686 as critical for BCL11b acetylation and nuclear localization, affecting its transcriptional activity and interactions with p300.

This study shows that BCL11B/CTIP2 regulates the TLR3-mediated cellular response to viral infections by inhibiting NEAT1 expression and dampening interferon production, thereby reinforcing HIV-1 latency.

The paper identifies RNA and protein interactors of BCL11b, showing its role in RNA processing and disease pathways.

This study shows that KAP1 represses HIV-1 gene expression in myeloid cells by interacting with Tat and CTIP2, contributing to HIV-1 latency.

This study identifies new RNA polymerase III and II promoters in the Bovine Leukemia Virus genome, showing their role in miRNA expression and viral latency.