Author

Sarah M. Graff

Vanderbilt UniversityORCID 0000-0001-9305-1760Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Developmental Biology· 1 Genetics· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 1 Organism:human· 1 Organism:mouse· 1 Approach:statistical model· 1

Frequent Co-Authors

David Aaron JacobsoniD· 2 shared Prasanna K. DadiiD· 2 shared Aideen M McInerney-LeoiD· 1 shared Ariel S. Thorson· 1 shared Arya Y. NakheiD· 1 shared Charles M. SchaubiD· 1 shared Emma L. DuncaniD· 1 shared Gautami Amarnath· 1 shared

Activity by Year

2020-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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2 shared papersDevelopmental Biology

David Aaron JacobsonORCID

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2 papers

Recent·Most cited

Developmental BiologybioRxivPreprintJul 15, 2020

TRPM7 is a critical regulator of pancreatic endocrine development and high-fat diet-induced β-cell proliferation.

Molly K. Altman, Charles M. Schaub, Matthew T. Dickerson…·David Aaron Jacobson

This paper identifies TRPM7's critical role in pancreatic progenitor expansion and β-cell proliferation under insulin-resistant conditions through genetic studies in mice.

DOI 10.1101/2020.07.15.204974

mouseexperimental assay

𝕏 PostBluesky

GeneticsbioRxivPreprintFeb 4, 2020

A novel mutation in KCNK16 causing a gain-of-function in the TALK-1 potassium channel: a new cause of maturity onset diabetes of the young.

Sarah M. Graff, Stephanie R. Johnson, Paul J. Leo…·Emma L. Duncan

The paper identifies a novel gain-of-function mutation in KCNK16 causing MODY by impairing Ca2+ influx and glucose-stimulated insulin secretion.

DOI 10.1101/2020.02.04.929430

humanstatistical model

𝕏 PostBluesky