Author

Annika Forsingdal

Lundbeck (Denmark)ORCID 0000-0001-9480-4365Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Bioinformatics· 1 Genetic & Genomic Medicine· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Organism:mouse· 1 Assay:whole-exome sequencing· 1

Frequent Co-Authors

Jacob NielseniD· 2 shared Michael DidrikseniD· 2 shared Aaron GordoniD· 1 shared Allison M Britt· 1 shared Daniel H. GeschwindiD· 1 shared Dawn M. FilmyeriD· 1 shared Gabriel Lázaro‐MuñoziD· 1 shared Ib Vestergaard Klewe· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersGenetic & Genomic Medicine

Jacob NielsenORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Genetic & Genomic MedicineSchizophrenia BulletinPeer-reviewedOct 25, 2022

Increased Prevalence of Rare Copy Number Variants in Treatment-Resistant Psychosis

Martilias S. Farrell, Tyler E Dietterich, Matthew K. Harner…·Richard C. Josiassen

The paper finds that rare copy number variants (CNVs) are more common in treatment-resistant psychosis patients compared to a general schizophrenia cohort, with duplications at 15q11.2-q13.1 and 16p11.2 linked to treatment resistance.

32 citationstop 5%

humanwhole-exome sequencing

𝕏 PostBluesky

BioinformaticsMolecular PsychiatryPeer-reviewedNov 8, 2019

Transcriptomic networks implicate neuronal energetic abnormalities in three mouse models harboring autism and schizophrenia-associated mutations

Aaron Gordon, Annika Forsingdal, Ib Vestergaard Klewe…·Daniel H. Geschwind

The paper identifies shared gene expression modules in mouse models of autism and schizophrenia that link to neuronal energetic dysfunction.

35 citations

mousebulk RNA-seq

𝕏 PostBluesky