Author

Esther Redín

Memorial Sloan Kettering Cancer CenterORCID 0000-0001-9539-7298Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 2 Genomics· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Approach:statistical model· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:clustering· 1

Frequent Co-Authors

Charles M. RudiniD· 3 shared Àlvaro Quintanal-VillalongaiD· 2 shared Abhijit ParoliaiD· 1 shared Alister P. W. FunnelliD· 1 shared Andrea ArfèiD· 1 shared Andrea VenturaiD· 1 shared Andrew McPhersoniD· 1 shared Arielle ElkriefiD· 1 shared

Activity by Year

2024-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 22 mutual co-author links

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3 shared papersCancer Biology

Charles M. RudinORCID

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3 papers

Recent·Most cited

Cancer BiologyTranslational OncologyPeer-reviewedJul 20, 2026

SMARCA4, STK11, and KEAP1 co-inactivation associates with poor prognosis and upregulation of the TGF-β pathway in lung adenocarcinoma

Emily A. Costa, Barbara Vaz de Mello, Corrin A. Wohlhieter…·Charles M. Rudin

The paper identifies SMARCA4/STK11/KEAP1 triple mutant lung adenocarcinoma as a poor prognosis subset and links it to TGF-β pathway activation.

DOI 10.1016/j.tranon.2026.102915

humanbulk RNA-seq

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GenomicsbioRxivPreprintApr 30, 2026

Evolution of oncogene amplification across 86,000 cancer cell genomes

Jake June-Koo Lee, Sohrab Salehi, Matthew A. Myers…·Sohrab P. Shah

The paper analyzes the evolutionary trajectories of high-level copy-number amplifications in cancer cell genomes to distinguish between extrachromosomal and intrachromosomal amplification mechanisms.

2 citations

humanwhole-genome sequencing

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Cancer BiologyCancer CellPeer-reviewedJul 18, 2024

Targeting the mSWI/SNF Complex in POU2F-POU2AF Transcription Factor-Driven Malignancies

Tongchen He, Lanbo Xiao, Yuanyuan Qiao…·Arul M. Chinnaiyan

This study identifies a therapeutic vulnerability in POU2F-POU2AF-driven cancers by showing that targeting the mSWI/SNF complex with a PROTAC degrader reduces tumor growth in preclinical models.

63 citationstop 1%

humanbulk RNA-seq

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