Author

Beate Henrichfreise

University of BonnORCID 0000-0001-9783-8157Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Organism:chicken / avian· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

George W. LiechtiiD· 2 shared Iris LöckeneriD· 2 shared Aissata Diallo· 1 shared Anna KlöckneriD· 1 shared Christian F. OtteniD· 1 shared Grace Overman· 1 shared Josie Blair· 1 shared Jula Reuter· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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2 shared papersMicrobiology

George W. LiechtiORCID

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2 papers

Recent·Most cited

Characterization of the Cell Division-Associated Peptidoglycan Amidase AmiA of Chlamydia trachomatis

Julia Dannenberg, Junghoon Lee, George W. Liechti, Christian F. Otten, Iris Löckener, Jula Reuter, Anna Klöckner, Sebastian Krannich, Tanja Schneider, Scot P. Ouellette, Beate Henrichfreise

This study characterizes AmiA_Ct, a peptidoglycan amidase in Chlamydia trachomatis, showing its role in degrading peptidoglycan during cell division and supporting normal growth and development.

Journal of Bacteriology · Mar 11, 2026Peer-reviewedMicrobiology

Chlamydia trachomatis restricts signaling through NOD2 until late in the pathogen's developmental cycle

Grace Overman, Iris Löckener, Zachary Williford, Sung Davis, Aissata Diallo, Josie Blair, Beate Henrichfreise, George W. Liechti

This study shows Chlamydia trachomatis delays NOD2 signaling until late in its developmental cycle, possibly to avoid immune detection, and suggests PG degradation reduces NOD2 ligand generation.

Infection and Immunity · Dec 19, 2025Peer-reviewedMicrobiology