Christopher C. Quinn

UNC-16 interacts with LRK-1 and WDFY-3 to regulate the termination of axon growth.

This study identifies UNC-16's role in axon termination through interactions with LRK-1 and WDFY-3, highlighting the endolysosomal system's involvement.

Autism candidate gene rbm-26(RBM26/27) regulates MALS-1 to protect against mitochondrial dysfunction and axon degeneration during neurodevelopment.

This study identifies RBM-26 as a regulator of MALS-1 that prevents mitochondrial dysfunction and axon degeneration during neurodevelopment in C. elegans.

An autism-associated calcium channel variant causes defects in neuronal polarity and axon termination in the ALM neuron of C. elegans

This study shows that an autism-associated CACNA1C variant disrupts neuronal polarity and axon termination in C. elegans ALM neurons.