Author

Tamjid A. Chowdhury

University of Wisconsin–MilwaukeeORCID 0000-0001-5051-7030Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:C. elegans· 1

Frequent Co-Authors

Christopher C. QuinniD· 2 shared Amy SY Lee· 1 shared Cody J. DrozdiD· 1 shared David A Luy· 1 shared Dorian FaracheiD· 1 shared Garrett Scapellato· 1 shared

Activity by Year

2023-2024 · peak 1/yr

Co-Author Collaboration Network

6 top collaborators · 11 mutual co-author links

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2 shared papersNeuroscience

Christopher C. QuinnORCID

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2 papers

Recent·Most cited

NeuroscienceGeneticsPeer-reviewedApr 6, 2024

UNC-16 interacts with LRK-1 and WDFY-3 to regulate the termination of axon growth.

Cody J. Drozd, Tamjid A. Chowdhury, Christopher C. Quinn

This study identifies UNC-16's role in axon termination through interactions with LRK-1 and WDFY-3, highlighting the endolysosomal system's involvement.

4 citations

experimental assay

𝕏 PostBluesky

NeurosciencebioRxivPreprintOct 12, 2023

Autism candidate gene rbm-26(RBM26/27) regulates MALS-1 to protect against mitochondrial dysfunction and axon degeneration during neurodevelopment.

Tamjid A. Chowdhury, David A Luy, Garrett Scapellato…·Christopher C. Quinn

This study identifies RBM-26 as a regulator of MALS-1 that prevents mitochondrial dysfunction and axon degeneration during neurodevelopment in C. elegans.

1 citation

C. elegansexperimental assay

𝕏 PostBluesky