Author

Sheila A. Baker

University of IowaORCID 0000-0002-0259-0652Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell type annotation· 2 Assay:electronic health records· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1

Frequent Co-Authors

Colten K. LankfordiD· 2 shared Deepak Poria· 2 shared Eduardo C. SolessioiD· 2 shared Joseph G. LairdiD· 2 shared Shivangi M. Inamdar· 2 shared Vladimir J. KefaloviD· 2 shared Alex R Bock· 1 shared Bruce A. BerkowitziD· 1 shared

Activity by Year

2021-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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2 shared papersNeuroscience

Colten K. LankfordORCID

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3 papers

Recent·Most cited

Biomarkers indicate altered outer retinal fluid homeostasis in the absence of Kv2.1/Kv8.2 channels.

Joseph G. Laird, James Soetedjo, Shivangi M. Inamdar, Alex R Bock, Ehimenmen S. Ataman, Miles A. Pufall, Bruce A. Berkowitz, Sheila A. Baker

The paper investigates the role of Kv2.1/Kv8.2 channels in outer retinal fluid homeostasis using mouse models and finds altered fluid dynamics and upregulated osmosensitive genes in their absence.

bioRxiv · Jul 2, 2026 · PreprintNeuroscience

Cone-driven retinal responses are shaped by rod but not cone HCN1

Colten K. Lankford, Yumiko Umino, Deepak Poria, Vladimir J. Kefalov, Eduardo C. Solessio, Sheila A. Baker

The paper investigates the role of HCN1 channels in retinal signaling, finding that rod-driven responses are affected by HCN1 knockout, while cone-driven responses remain unchanged.

Journal of Neuroscience · Apr 18, 2022Peer-reviewedNeuroscience

Differential impact of Kv8.2 loss on rod and cone signaling and degeneration.

Shivangi M. Inamdar, Colten K. Lankford, Deepak Poria, Joseph G. Laird, Eduardo C. Solessio, Vladimir J. Kefalov, Sheila A. Baker

This study examines the impact of Kv8.2 loss on rod and cone photoreceptor signaling and degeneration using a knockout mouse model.

Human Molecular Genetics · Oct 12, 2021Peer-reviewedNeuroscience