HSP40 overexpression in pacemaker neurons protects against circadian dysfunction in a Drosophila model of Huntington's Disease
Pavitra Prakash, Arpit Kumar Pradhan, Vasu Sheeba
This study shows that overexpression of HSP40 in pacemaker neurons rescues circadian dysfunction in a Drosophila Huntington's Disease model by restoring circadian proteins and reducing Huntingtin inclusion load.
Disease Models & Mechanisms · May 30, 2022Peer-reviewedNeuroscience