Author

Hailun Li

Hôpital Necker-Enfants MaladesORCID 0000-0002-0764-2837Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Research Focus:GWAS / association mapping· 1 Organism:human· 1 Assay:scRNA-seq· 1 Approach:statistical model· 1

Frequent Co-Authors

Ana Esteve‐SoléiD· 1 shared Andrés Augusto AriasiD· 1 shared Andrés BaenaiD· 1 shared Aniss RafikiD· 1 shared Anne PueliD· 1 shared Antoni Noguera‐JuliániD· 1 shared Aurélie CobatiD· 1 shared Ayça Aslan KıykımiD· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperGenetics

Ana Esteve‐SoléORCID

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1 paper

Recent·Most cited

Humans homozygous for rare or common hypomorphic IL23R variants are prone to tuberculosis

Diana Olguín-Calderón, Laura E. Kilpatrick, Clément Conil, Quentin Philippot, Masato Ogishi, Joseph Vellutini, Ji Eun Han, Narelle Keating, Hailun Li, Geetha Rao, Jonathan Bohlen, Charles S. Lay, Simon Platt, Gaspard Kerner, Elsa Feredj, , Stéphanie Boisson‐Dupuis

Homozygosity for hypomorphic IL23R variants (G149R, G300V, R381Q) partly impairs IL-23-dependent IFN-γ production in patient cells; with L372F (effect inferred), they are enriched in tuberculosis, not MSMD or CMC patients.

The Journal of Experimental Medicine · Jul 9, 2026Peer-reviewedGenetics✦ In-depth digest