Author

Anita Göndör

Karolinska University HospitalORCID 0000-0002-1523-4411Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1 Genomics· 1

Signature Assays, Organisms & Methods

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Research Focus:gene regulatory network inference· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Assay:ChIP-seq· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1

Frequent Co-Authors

Barbara A. ScholziD· 2 shared Narsis A. KianiiD· 2 shared Rolf I. OHLSSONiD· 2 shared Alejandro Fernandez-WoodbridgeiD· 1 shared Anne Hansen ReeiD· 1 shared Ayushi ChaurasiyaiD· 1 shared Chenying GaoiD· 1 shared David Gómez-CabreroiD· 1 shared

Activity by Year

2018-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersCancer Biology

Barbara A. ScholzORCID

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2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintJan 31, 2026

A distal CTCF-binding site drives MYC expression plasticity in a negative feed-forward loop

Chenying Gao, Mirco Martino, Ayushi Chaurasiya…·Anita Göndör

The paper identifies a CTCF-binding site that regulates MYC expression plasticity through a negative feed-forward loop involving CCAT1 eRNA and MYC protein feedback.

DOI 10.64898/2026.01.28.702300

humanbulk RNA-seq

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GenomicsbioRxivPreprintMar 27, 2018

The ultra-sensitive Nodewalk technique identifies stochastic from virtual, population-based enhancer hubs regulating MYC in 3D: Implications for the fitness of cancer cells

Noriyuki Sumida, Emmanouil G Sifakis, Barbara A. Scholz…·Rolf I. OHLSSON

The paper introduces Nodewalk, a highly sensitive method to analyze chromatin networks, revealing that enhancers around MYC operate in a stochastic, mutually exclusive manner, impacting cancer cell fitness.

5 citations

ChIP-seqexperimental assay

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