Author

Thomas Brickler

Stanford MedicineORCID 0000-0002-1580-4808Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1

Frequent Co-Authors

Allison M. Banuelos· 1 shared Anna ShcherbinaiD· 1 shared Cyndhavi NarayananiD· 1 shared Irving L. WeissmaniD· 1 shared Jing Tan BianiD· 1 shared Jingling Li· 1 shared Kristopher David Marjon· 1 shared Sravani BanerjeeiD· 1 shared

Activity by Year

2021-2021 · peak 1/yr

Co-Author Collaboration Network

9 top collaborators · 36 mutual co-author links

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1 shared paperNeuroscience

Allison M. Banuelos

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1 paper

Recent·Most cited

NeuroscienceProceedings of the National Academy of SciencesPeer-reviewedApr 8, 2021

Overexpression of CD47 is associated with brain overgrowth in 16p11.2 deletion syndrome

Jingling Li, Thomas Brickler, Allison M. Banuelos…·Sundari Chetty

The paper investigates the role of CD47 overexpression in 16p11.2 deletion syndrome, showing it contributes to brain overgrowth by inhibiting phagocytosis in neural progenitor and oligodendrocyte progenitor cells.

38 citations

humanexperimental assay

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