Author

Rita Zilhão

University of LisbonORCID 0000-0002-1885-5432Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Cancer Biology· 1 Developmental Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

Ana Rita CarlosiD· 2 shared Diogo R. FernandesiD· 2 shared Albena T. Dinkova‐KostovaiD· 1 shared Ana Beatriz MachadoiD· 1 shared Bérénice Saget· 1 shared Catarina M. AzevedoiD· 1 shared Catarina Melo· 1 shared Cátia Rebelo de AlmeidaiD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCancer Biology

Ana Rita CarlosORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintAug 26, 2026

CD24 Acts as an Evolutionarily Conserved Innate Immune Checkpoint in Colorectal Cancer

Ana Beatriz Machado, Cátia Rebelo de Almeida, Catarina M. Azevedo…·Rita Fior

This study identifies CD24 as an innate immune checkpoint in colorectal cancer that promotes tumor survival by inhibiting macrophage-mediated clearance, validated through zebrafish models and human data.

DOI 10.64898/2026.08.25.746757

zebrafishproteomics

Developmental BiologyLife Science AlliancePeer-reviewedOct 8, 2024

Deregulation of multiple mechanisms shapes the onset of LAMA2-congenital muscular dystrophy

Susana G. Martins, Vanessa Ribeiro, Catarina Melo…·Sólveig Þorsteinsdóttir

This study investigates the early molecular changes in LAMA2-CMD using a mouse model, identifying gene expression, cytoskeletal, and DNA repair defects at disease onset.

14 citations

mousebulk RNA-seq