Author

Darryl Hayward

The Francis Crick InstituteORCID 0000-0002-1927-8921Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Developmental Biology· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:imaging· 1 Approach:statistical model· 1

Frequent Co-Authors

Dorota Gibbins· 2 shared Elizabeth FisheriD· 2 shared Victor L. J. TybulewicziD· 2 shared Ambrosius P. SnijdersiD· 1 shared André StrydomiD· 1 shared Bernard M. SiowiD· 1 shared Callan L BuechsenschuetziD· 1 shared Camilla SimmonsiD· 1 shared

Activity by Year

2023-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersNeuroscience

Dorota Gibbins

Mutual co-authors in cluster

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2 papers

Recent·Most cited

NeuroscienceNeurobiology of DiseasePeer-reviewedOct 29, 2023

Investigating Brain Alterations in the Dp1Tyb Mouse Model of Down Syndrome

Maria Elisa Serrano Navacerrada, Eugene Kim, Bernard M. Siow…·Diana Cash

This study examines the brain phenotype of Dp1Tyb mice, a Down syndrome model, revealing neuroanatomical and biochemical alterations compared to wild-type controls.

7 citations

mouseimaging

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Developmental BiologybioRxivPreprintSep 18, 2023

Congenital heart defects in Down syndrome are caused by increased dosage of DYRK1A

Eva Lana‐Elola, Rifdat Aoidi, Miriam Llorian…·Victor L. J. Tybulewicz

The paper shows increased DYRK1A dosage causes mitochondrial dysfunction and congenital heart defects in Down syndrome, which can be mitigated by a DYRK1A inhibitor.

DOI 10.1101/2023.09.18.558244

humanmouse

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