Author

Tamás Bozó

Semmelweis UniversityORCID 0000-0002-2643-0661Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1

Frequent Co-Authors

Éva GráczeriD· 2 shared Katalin PásztyiD· 2 shared Miklós KellermayeriD· 2 shared Nataliya Di DonatoiD· 2 shared Á. AltorjayiD· 1 shared Andrea VargaiD· 1 shared Andréa VargaiD· 1 shared Elena BattirossiiD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 40 mutual co-author links

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2 shared papersBiophysics

Éva GráczerORCID

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2 papers

Recent·Most cited

BiophysicsEuropean Journal of Cell BiologyPeer-reviewedOct 9, 2026

Non-muscle actinopathy-associated loss-of-function actin variants modulate cytoskeletal reorganization

Éva Gráczer, Kira Dakos, Tamás Bozó…·Andrea Varga

This study examines how specific ACTB gene variants affect cytoskeletal reorganization in fibroblasts, finding that G302A reduces cell stiffness and alters cofilin and myosin light chain phosphorylation.

1 citation

humanproteomics

𝕏 PostBluesky

BiophysicsThe FASEB JournalPeer-reviewedJan 5, 2026

The Baraitser-Winter Cerebrofrontofacial Syndrome recurrent R196H variant in cytoplasmic β-actin impairs its cellular polymerization and stability

Éva Gráczer, Elena Battirossi, Tamás Bozó…·Andréa Varga

This study investigates the R196H mutation in β-actin, showing it reduces F-actin content and cell stiffness, weakens actin-membrane attachment, and impairs cofilin interaction.

3 citationstop 3%

humanproteomics

𝕏 PostBluesky