Author

Andrew B. Caldwell

University of California San DiegoORCID 0000-0002-2850-7677Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2 Genetics· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 3 Organism:human· 3 Approach:statistical model· 3 Assay:ATAC-seq· 2 Assay:bulk RNA-seq· 2 Research Focus:gene regulatory network inference· 2 Assay:ChIP-seq· 1 Approach:experimental assay· 1

Frequent Co-Authors

Shankar SubramaniamiD· 3 shared Gabriel George HaddadiD· 2 shared Dan ZhouiD· 1 shared Dan ZhouiD· 1 shared Douglas R. Galasko· 1 shared Francisco C. VillafuerteiD· 1 shared Gary P. SchrothiD· 1 shared Ila Dwivedi· 1 shared

Activity by Year

2020-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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3 shared papersNeuroscience

Shankar SubramaniamORCID

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3 papers

Recent·Most cited

GeneticsbioRxivPreprintOct 20, 2025

Integrated multiomics reveals inflammation-driven excessive erythrocytosis in subjects with Monge's disease

Priti Azad, Andrew B. Caldwell, Francisco C. Villafuerte…·Gabriel George Haddad

This study uses multiomics analysis to show that inflammation-driven NFκB activation under hypoxia causes excessive erythrocytosis in Monge's disease.

DOI 10.1101/2025.10.20.683487

humanbulk RNA-seq

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NeuroscienceTranslational PsychiatryPeer-reviewedMay 5, 2023

Methadone alters transcriptional programs associated with synapse formation in human cortical organoids

Ila Dwivedi, Andrew B. Caldwell, Dan Zhou…·Gabriel George Haddad

This study shows methadone exposure during early cortical development alters synapse-related transcriptional programs in human cortical organoids.

13 citationstop 5%

humanbulk RNA-seq

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NeuroscienceScience AdvancesPeer-reviewedNov 13, 2020

Dedifferentiation and neuronal repression define Familial Alzheimer's Disease

Andrew B. Caldwell, Qing Liu, Gary P. Schroth…·Shankar Subramaniam

This study identifies dedifferentiation and neuronal repression as key mechanisms in Familial Alzheimer's Disease using patient-derived neurons and multi-omics analysis.

69 citationstop 2%

humanscRNA-seq

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