Author

Neyaz Alam

Chittaranjan National Cancer InstituteORCID 0000-0002-2941-6769Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Organism:mouse· 1

Frequent Co-Authors

Aishwarya GuhaiD· 2 shared Anamika BoseiD· 2 shared Anirban SarkariD· 2 shared Jasmine SultanaiD· 2 shared Juhina DasiD· 2 shared Mohona ChakravartiiD· 2 shared Nilanjan GangulyiD· 2 shared Pritha Roy ChoudhuryiD· 2 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersCancer Biology

Aishwarya GuhaORCID

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2 papers

Recent·Most cited

Breast cancer stem cells mediated CD8+ T cell exhaustion among different molecular subtypes of breast cancer regulated via NOTCH1/RBPJ/PD-L1 axis

Jasmine Sultana, Aishwarya Guha, Mohona Chakravarti, Goutam Ulgekar, Saurav Bera, Pritha Roy Choudhury, Sukanya Dhar, Juhina Das, Nilanjan Ganguly, Anirban Sarkar, Mariya Saify, Monika Rana, Prodipto Das, Akata Saha, Nirmalya Ganguli, , Saptak Banerjee

This study identifies BCSCs as drivers of CD8+ T cell exhaustion in breast cancer subtypes, showing NOTCH1/RBPJ/PD-L1 axis regulation and potential therapeutic targeting.

bioRxiv · May 26, 2026 · PreprintCancer Biology

Tumor-Educated-Platelets interact with Breast Cancer-Stem-Cells via P-selectin- PSGL1 and ensure stemness and metastasis through WNT-β-Catenin-VEGF-VEGFR2 intra-cellular signaling: Therapeutic modulation by aspirin

Aishwarya Guha, Jasmine Sultana, Avishek Bhuniya, Mohona Chakravarti, Saurav Bera, Anirban Sarkar, Sukanya Dhar, Pritha Roy Choudhury, Prodipto Das, Juhina Das, Nilanjan Ganguly, Ipsita Guha, Dhrubajyoti Bairagya, Tapasi Das, Neyaz Alam, , Saptak Banerjee

This study identifies tumor-educated platelets (TEPs) interacting with breast cancer stem cells (BCSCs) via P-selectin-PSGL1 to promote stemness and metastasis through WNT-β-Catenin-VEGF-VEGFR2 signaling, and shows aspirin can reverse this effect.

Breast Cancer Research · Dec 15, 2025Peer-reviewedCancer Biology