Author

Anamika Bose

National Institute of Pharmaceutical Education and ResearchORCID 0000-0003-2241-2033Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Assay:bulk RNA-seq· 2 Research Focus:cancer & tumor microenvironment· 2 Organism:human· 2 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1

Frequent Co-Authors

Anirban SarkariD· 3 shared Jasmine SultanaiD· 3 shared Juhina DasiD· 3 shared Mohona ChakravartiiD· 3 shared Pritha Roy ChoudhuryiD· 3 shared Rathindranath BaraliD· 3 shared Saptak BanerjeeiD· 3 shared Saurav BeraiD· 3 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersCancer Biology

Anirban SarkarORCID

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3 papers

Recent·Most cited

Cancer BiologyThe Journal of ImmunologyPeer-reviewedOct 1, 2026

Experimental type 1 diabetes metabolically rejuvenates CD8+ T cells for improved control of tumor growth through an IGF1-IGF1R axis

Anirban Sarkar, Sukanya Dhar, Saurav Bera…·Anamika Bose

This study shows that experimental type 1 diabetes enhances CD8+ T cell function via the IGF1-IGF1R axis, leading to better tumor control.

DOI 10.1093/jimmun/vkag262

mouseproteomics

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Cancer BiologybioRxivPreprintMay 26, 2026

Breast cancer stem cells mediated CD8+ T cell exhaustion among different molecular subtypes of breast cancer regulated via NOTCH1/RBPJ/PD-L1 axis

Jasmine Sultana, Aishwarya Guha, Mohona Chakravarti…·Saptak Banerjee

This study identifies BCSCs as drivers of CD8+ T cell exhaustion in breast cancer subtypes, showing NOTCH1/RBPJ/PD-L1 axis regulation and potential therapeutic targeting.

DOI 10.64898/2026.05.22.727059

humanbulk RNA-seq

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Cancer BiologyBreast Cancer ResearchPeer-reviewedDec 15, 2025

Tumor-Educated-Platelets interact with Breast Cancer-Stem-Cells via P-selectin- PSGL1 and ensure stemness and metastasis through WNT-β-Catenin-VEGF-VEGFR2 intra-cellular signaling: Therapeutic modulation by aspirin

Aishwarya Guha, Jasmine Sultana, Avishek Bhuniya…·Saptak Banerjee

This study identifies tumor-educated platelets (TEPs) interacting with breast cancer stem cells (BCSCs) via P-selectin-PSGL1 to promote stemness and metastasis through WNT-β-Catenin-VEGF-VEGFR2 signaling, and shows aspirin can reverse this effect.

5 citations

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