Author

Gamze Yelgen

Üsküdar UniversityORCID 0000-0002-3168-835XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Assay:CRISPR screen· 2 Research Focus:drug response / drug discovery· 2 Organism:bacteria (other)· 1 Research Focus:benchmarking· 1 Assay:proteomics· 1

Frequent Co-Authors

Cihan TaştaniD· 3 shared Sibel Pınar OdabaşiD· 3 shared Berranur SertiD· 2 shared Enes BaliD· 2 shared Gamze GüldeniD· 2 shared Hasret AraziD· 2 shared Ilayda CavrariD· 2 shared Nulifer Neslihan TiryakiiD· 2 shared

Activity by Year

2022-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersGenetics

Cihan TaştanORCID

Mutual co-authors in cluster

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3 papers

Recent·Most cited

GeneticsbioRxivPreprintAug 21, 2023

Enhancing In Vitro SMN Protein Expression and Cell Viability through Xeno-Nucleic Acid-Based ASOs in Spinal Muscular Atrophy

Ozum Kilic, Hale Ahsen Babar, Cemre Can Inci…·Cihan Taştan

This study evaluates XNA-DNA-ASO sequences for enhancing SMN protein expression and cell viability in SMA fibroblasts, showing improved stability and reduced toxicity compared to conventional ASOs.

DOI 10.1101/2023.08.21.552929

humanexperimental assay

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Molecular BiologybioRxivPreprintJul 22, 2022

Autonomous Remotely Controlled Closed System Transgenic Cell Technologies Robot: CRISPR.BOT

Fatmanur Erkek, Gamze Gülden, Berranur Sert…·Cihan Taştan

This paper introduces CRISPR.BOT, a robotic system for automated genetic engineering tasks, achieving high-purity single-cell subcloning and reducing labor and costs in biosafety level 3 experiments.

DOI 10.1101/2022.07.22.497959

humanbacteria (other)

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GeneticsbioRxivPreprintMar 21, 2022

Exon7 Targeted CRISPR-Prime Editing Approaches for SMN2 Gene Editing in Spinal Muscular Atrophy (SMA) Disease Can Increase In Vitro SMN Expression

Sibel Pınar Odabaş, Enes Bal, Gamze Yelgen…·Cihan Taştan

This study uses CRISPR-Prime Editing to correct two point mutations in the SMN2 gene, increasing SMN expression in SMA-related cells.

4 citations

humanCRISPR screen

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