Author

Giuseppina Tesco

Tufts UniversityORCID 0000-0002-3336-7488Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Organism:human· 1 Organism:mouse· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

Philip G. HaydoniD· 2 shared Alice BragaiD· 1 shared Beatrice MenicacciiD· 1 shared David Alan BennettiD· 1 shared David Lee KaplaniD· 1 shared Gregory W. CarteriD· 1 shared Mattia BonzanniiD· 1 shared Nicolas RouleauiD· 1 shared

Activity by Year

2023-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersNeuroscience

Philip G. HaydonORCID

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2 papers

Recent·Most cited

Adenosine deficiency facilitates CA1 synaptic hyperexcitability in the presymptomatic phase of a knock in mouse model of Alzheimer's disease.

Mattia Bonzanni, Alice Braga, Takashi Saito, Takaomi C. Saido, Giuseppina Tesco, Philip G. Haydon

The paper shows that adenosine deficiency in a mouse model of Alzheimer's disease leads to synaptic hyperexcitability during the asymptomatic phase, which can be restored by pharmacologic or dietary interventions.

bioRxiv · Apr 24, 2024 · PreprintNeuroscience

3D bioengineered neural tissue generated from patient-derived iPSCs develops time-dependent phenotypes and transcriptional features of Alzheimer's disease

Selene Lomoio, Ravi S. Pandey, Nicolas Rouleau, Beatrice Menicacci, WonHee Kim, William L. Cantley, Philip G. Haydon, David Alan Bennett, Tracy L. Young‐Pearse, Gregory W. Carter, David Lee Kaplan, Giuseppina Tesco

Molecular Psychiatry · Jun 26, 2023Peer-reviewedNeuroscience