Author

Mohammad Haddadi

Zabol UniversityORCID 0000-0002-3374-0879Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Genetic & Genomic Medicine· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Organism:Drosophila· 1 Approach:experimental assay· 1 Organism:human· 1 Approach:statistical model· 1

Frequent Co-Authors

David Isaac FinkelsteiniD· 1 shared S. R. Ramesh· 1 shared Samaneh Reiszadeh JahromiiD· 1 shared Yasaman Mostaghimi· 1 shared Zohreh Hojjati· 1 shared

Activity by Year

2020-2025 · peak 1/yr

Co-Author Collaboration Network

5 top collaborators · 4 mutual co-author links

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1 shared paperNeuroscience

David Isaac FinkelsteinORCID

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2 papers

Recent·Most cited

LncRNA PAN3-AS1 modulates cilium assemble signaling pathway through regulation of RPGR as a potential MS diagnostic biomarker: integrated systems biology investigation

Yasaman Mostaghimi, Mohammad Haddadi, Zohreh Hojjati

The paper identifies PAN3-AS1 as a low-expressed lncRNA in MS samples that regulates RPGR, potentially serving as a diagnostic biomarker through its role in cilium assembly and autophagy.

Journal of Molecular Neuroscience · Apr 14, 2025Peer-reviewedGenetic & Genomic Medicine

SNCAE46K transgenic Drosophila Model of Parkinsons Disease Confirmed the Causative Role of Oxidative Stress

Samaneh Reiszadeh Jahromi, S. R. Ramesh, David Isaac Finkelstein, Mohammad Haddadi

This study creates a Drosophila model overexpressing the E46K mutant SNCA gene, confirming its role in inducing oxidative stress and neurodegeneration associated with Parkinson's disease.

bioRxiv · Feb 28, 2020 · PreprintNeuroscience