Author

Alberto de la Fuente

Hamad bin Khalifa UniversityORCID 0000-0002-3697-3635Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:human· 2 Research Focus:differential expression· 1 Assay:electronic health records· 1 Approach:experimental assay· 1 Approach:machine learning (classical)· 1 Assay:proteomics· 1

Frequent Co-Authors

Bermet Abylova· 1 shared Diana Anuar· 1 shared Gowher AliiD· 1 shared Houari AbdesselemiD· 1 shared Houda Yasmine Ali MoussaiD· 1 shared Hyung‐Goo KimiD· 1 shared Ilham Bensmail· 1 shared Ioannis Nikolaos PetropoulosiD· 1 shared

Activity by Year

2022-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperNeuroscience

Bermet Abylova

Mutual co-authors in cluster

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2 papers

Recent·Most cited

A New Serological Autoantibody Signature Associated with Multiple Sclerosis

Houari Abdesselem, Alberto de la Fuente, Ilham Bensmail, Israa Elbashir, Tan Ti-Myen, Bermet Abylova, Lay Koon, Diana Anuar, Jonathan Michael Blackburn, Rayaz Ahmed Malik, Ioannis Nikolaos Petropoulos

This study identifies a serological autoantibody signature in MS patients, highlighting MX1 autoantibodies as a potential biomarker linked to antiviral immune responses.

Neurobiology of Disease · Oct 1, 2025Peer-reviewedNeuroscience

Deletion of TRPC6, an autism risk gene, induces hyperexcitability in cortical neurons derived from human pluripotent stem cells

Kyung Chul Shin, Gowher Ali, Houda Yasmine Ali Moussa, Vijay R. Gupta, Alberto de la Fuente, Hyung‐Goo Kim, Lawrence Walter Stanton, Yongsoo Park

This study shows that deleting TRPC6 in human cortical neurons increases neuronal hyperexcitability by reducing SOCE signaling.

bioRxiv · Nov 14, 2022 · PreprintNeuroscience