Author

Vance V. Soares

Johns Hopkins UniversityORCID 0000-0002-4094-3316Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Organism:mouse· 1 Approach:statistical model· 1

Frequent Co-Authors

Joshua C. DoloffiD· 2 shared Annie E. Newell‐FugateiD· 1 shared Calvin R. SchusteriD· 1 shared Christine Guo LianiD· 1 shared Devin CooniD· 1 shared Erik ReicheiD· 1 shared Hunter Hoover‐Watson· 1 shared J Crawford Daniel· 1 shared

Activity by Year

2024-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersBioengineering

Joshua C. DoloffORCID

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2 papers

Recent·Most cited

Immunologic comparisons of strain and induction method in an improved mouse model of intrauterine fibrosis

Jamie L. Hernandez, J Crawford Daniel, Jessica L. Stelzel, Neeti R. Prasad, Vance V. Soares, Joshua C. Doloff

This study compares intrauterine fibrosis development and immune responses across mouse strains and induction methods to identify a more clinically relevant model.

bioRxiv · Oct 30, 2024 · PreprintBioengineering

Angiogenic Steroids Induce Pathologic Scarring in a Preclinical Swine Model Via Dysfunctional Extracellular Matrix Deposition.

Erik Reiche, Patrick R. Keller, Vance V. Soares, Calvin R. Schuster, Siti Rahmayanti, Jessica Mroueh, Vanessa Mroueh, Marie Billaud, Sophia Hu, Hunter Hoover‐Watson, Christine Guo Lian, Yu Tan, Joshua C. Doloff, Annie E. Newell‐Fugate, Devin Coon

This study shows testosterone increases fibrosis and scar strength in a swine model, suggesting androgenic steroids contribute to pathologic scarring through extracellular matrix changes.

The FASEB Journal · Mar 26, 2024Peer-reviewedSystems Biology