Author

Ying Li

University of ChicagoORCID 0000-0002-4107-5442Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Ying Li26 papers · Emory University

Biology Disciplines

Click to filter author papers

Genetics· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Organism:human· 2 Approach:statistical model· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Assay:electronic health records· 1 Research Focus:gene regulatory network inference· 1 Research Focus:variant calling· 1

Frequent Co-Authors

Abhinav SrinathiD· 1 shared Aditi Joshi· 1 shared Aileen A. ReniD· 1 shared Chang CheniD· 1 shared Daniel A. SnellingsiD· 1 shared Douglas A. MarchukiD· 1 shared Howard C. NusbaumiD· 1 shared Issam A. AwadiD· 1 shared

Activity by Year

2022-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperGenetics

Abhinav SrinathORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

NeurosciencebioRxivPreprintAug 20, 2026

Listening with your heart: The heartbeat shapes auditory object formation by suppressing the early neural response to sound

John P. Veillette, Aditi Joshi, Ying Li, Howard C. Nusbaum

The paper shows that suppressing early neural responses to sound during the cardiac cycle can lead to qualitative changes in auditory perception, such as perceiving sounds as separate streams.

DOI 10.64898/2026.08.17.745255

humanelectronic health records

GeneticsNature Cardiovascular ResearchPeer-reviewedMar 14, 2022

Developmental Venous Anomalies are a Genetic Primer for Cerebral Cavernous Malformations

Daniel A. Snellings, Romuald Girard, Rhonda Lightle…·Douglas A. Marchuk

The paper identifies PIK3CA mutations in developmental venous anomalies (DVA) and CCM, showing DVA act as a genetic primer for CCM through shared mutations.

52 citationstop 3%

humanDNA methylation