Author

Adele Woodhouse

University of TasmaniaORCID 0000-0002-4246-7624Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:mouse· 2 Research Focus:benchmarking· 1 Assay:ChIP-seq· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Approach:statistical model· 1

Frequent Co-Authors

Adam K. WalkeriD· 1 shared Andrew J. PhippsiD· 1 shared Catherine A. BlizzardiD· 1 shared Jake M. CashioniD· 1 shared Katherine A. GilesiD· 1 shared Katherine E. A. LewisiD· 1 shared Laura A. RealeiD· 1 shared Marcus S. DyeriD· 1 shared

Activity by Year

2020-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperNeuroscience

Adam K. WalkerORCID

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2 papers

Recent·Most cited

H3K4me3 enrichment defines neuronal age, while a youthful H3K27ac signature is recapitulated in aged neurons

Katherine A. Giles, Andrew J. Phipps, Jake M. Cashion, Shannon N. Huskins, Tim R. Mercer, Mark D. Robinson, Adele Woodhouse, Phillippa C. Taberlay

This study maps H3K4me3 and H3K27ac in mouse neurons across lifespan, revealing epigenetic changes associated with neuronal aging and identifying new aging-related features.

bioRxiv · Nov 11, 2021 · PreprintMolecular Biology

Amyotrophic lateral sclerosis associated mislocalisation of TDP-43 to the cytoplasm causes cortical hyperexcitability and reduced excitatory neurotransmission in the motor cortex.

Marcus S. Dyer, Laura A. Reale, Katherine E. A. Lewis, Adam K. Walker, Tracey C. Dickson, Adele Woodhouse, Catherine A. Blizzard

This study shows that TDP-43 mislocalization to the cytoplasm in motor cortex neurons causes hyperexcitability and reduced excitatory neurotransmission, linking early ALS pathology to TDP-43 mislocalization.

Journal of Neurochemistry · Oct 16, 2020Peer-reviewedNeuroscience