Author

Smitha Srinivasachar Badarinarayan

Cornell UniversityORCID 0000-0002-4591-0513Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Molecular Biology· 1 Systems Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Assay:bulk RNA-seq· 1 Assay:ChIP-seq· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Assay:scRNA-seq· 1 Approach:statistical model· 1 Approach:transformer / foundation model· 1

Frequent Co-Authors

Adrian T. GrzybowskiiD· 1 shared Ankit Ved AroraiD· 1 shared Daksha Munot· 1 shared Daniel SauteriD· 1 shared Jan Eric KolbergiD· 1 shared Julián NeñeriD· 1 shared Martin Müller· 1 shared Natalia SavytskaiD· 1 shared

Activity by Year

2023-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperSystems Biology

Adrian T. GrzybowskiORCID

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2 papers

Recent·Most cited

TRAILBLAZER: generative multicellular perturbation model of biology

Julián Neñer, Padmalochini Selvamani, Smitha Srinivasachar Badarinarayan, Naveen Chandramohan, Adrian T. Grzybowski

TRAILBLAZER predicts multicellular patient-level responses to interventions while preserving single-cell resolution and enabling generalization.

bioRxiv · Mar 23, 2026 · PreprintSystems Biology

SARS-CoV-2 infection activates endogenous retroviruses of the LTR69 subfamily

Ankit Ved Arora, Jan Eric Kolberg, Smitha Srinivasachar Badarinarayan, Natalia Savytska, Daksha Munot, Martin Müller, Veronika Krchlíková, Daniel Sauter, Vikas Bansal

This study shows SARS-CoV-2 infection activates the LTR69 subfamily of endogenous retroviruses, including Dup69, which influences gene expression near PTPRN2 and ENSG00000289418.

Mobile DNA · Sep 4, 2023Peer-reviewedMolecular Biology