You-Ming Chen

Mitigation of Aβ25-35-Induced Cognitive Deficits in C57BL/6 Mice via Thermal Cycling Stimulation Employing Focused Ultrasound

This study uses focused ultrasound-mediated thermal cycling stimulation to improve cognitive deficits in Alzheimer's disease mice, showing enhanced cognitive performance and increased expression of Aβ-degrading enzymes.

Thermal cycling stimulation via nasal inhalation attenuates Aβ25-35-induced cognitive deficits in C57BL/6 mice

This study investigates the effects of thermal cycling-stimulation via nasal inhalation on cognitive deficits in mice with Aβ25-35-induced Alzheimer's disease, finding improvements in cognitive performance and reduced Aβ accumulation.

Thermal cycling-hyperthermia attenuates rotenone-induced cell injury in SH-SY5Y cells through heat-activated mechanisms

This study shows that thermal cycling-hyperthermia reduces rotenone-induced cell injury in SH-SY5Y cells by inhibiting mitochondrial apoptosis, ROS accumulation, and protein aggregation through heat-activated pathways.

Combination of cycling hyperthermia and Echinacoside creates synergistic curing effect on pancreatic cancer PANC-1 cells

The paper shows that combining Echinacoside with thermal-cycling hyperthermia enhances the anti-cancer effect on PANC-1 cells by targeting multiple apoptotic pathways.

Thermal cycling-hyperthermia ameliorates cognitive impairment of intracerebroventricular Aβ25-35-induced Alzheimer's disease in C57BL/6 mice

This study shows that thermal cycling-hyperthermia (TC-HT) is more effective than continuous hyperthermia in improving cognitive impairment and reducing Aβ and neuroinflammation in an Alzheimer's disease mouse model.

Protection of high-frequency low-intensity pulsed electric fields and BDNF for SH-SY5Y cells against hydrogen peroxide-induced cell damage

The paper investigates the combined effect of high-frequency low-intensity pulsed electric fields and BDNF on protecting SH-SY5Y cells from hydrogen peroxide-induced damage, finding synergistic neuroprotective effects through increased p-EGFR and MAPK activation.