Robson Francisco Carvalho

The paper evaluates feature representations for predicting cancer-associated sORFs in non-coding RNA using traditional machine learning models and finds that k-mer features outperform genomic language model embeddings.

This study shows that liver-derived Yap1 activation during organ repair induces systemic catabolism and muscle loss, similar to cancer cachexia.

The paper identifies miR-155-5p-induced gene expression changes in muscle atrophy and links them to disease-related pathways in skeletal muscle disorders.

The paper identifies non-canonical mediators of SARS-CoV-2 infection in placental cells through transcriptomic analysis.

This study shows preventive exercise in rats with pulmonary arterial hypertension increases collagen I and myosin 7 mRNA expression, suggesting it modulates extracellular matrix and myosin gene expression.

The paper identifies TRIB3 as a potential therapeutic target for COVID-19 by linking its decreased expression in aged lungs to increased susceptibility to SARS-CoV-2 infection.

The paper analyzes the aging whole blood transcriptome to identify genes potentially involved in SARS-CoV-2 interaction, finding FASLG as a candidate for severe COVID-19 in older adults.