Author

Alexander G. Robling

Indiana University IndianapolisORCID 0000-0002-3989-4780Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Developmental Biology· 1 Genetics· 1 Molecular Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 4 Organism:mouse· 4 Research Focus:differential expression· 3 Research Focus:cell type annotation· 2 Research Focus:gene regulatory network inference· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

Adrianna N. Masterson· 1 shared Andrei Bonteanu· 1 shared Andris KronbergsiD· 1 shared April M. Hoggatt· 1 shared Bart O. WilliamsiD· 1 shared Bohdan PidhaynyyiD· 1 shared Casey J. Droscha· 1 shared Cassandra R. DiegeliD· 1 shared

Activity by Year

2017-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 10 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperMolecular Biology

Adrianna N. Masterson

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Hepatic Yap1 activates systemic catabolism and muscle loss during organ repair: evidence for a liver-derived common mechanism with cancer cachexia.

Tewfik Hamidi, Yanlin Jiang, Tyler P. Robinson, Andris Kronbergs, Ernie D. Au, Joseph E. Rupert, Xiaoling Zhong, Siangsin Lal, Tiffany W. Liang, Alexander G. Robling, April M. Hoggatt, Robson Francisco Carvalho, Sarah Santiloni Cury, Clark D. Wells, Teresa A. Zimmers, Leonidas G. Koniaris

This study shows that liver-derived Yap1 activation during organ repair induces systemic catabolism and muscle loss, similar to cancer cachexia.

bioRxiv · Apr 1, 2025 · PreprintPhysiology

Gabapentin Disrupts Binding of Perlecan to the α2δ1 Voltage Sensitive Calcium Channel Subunit and Impairs Skeletal Mechanosensation

Perla Citlali Reyes Fernandez, Christian S. Wright, Adrianna N. Masterson, Xin Yi, Tristen V. Tellman, Andrei Bonteanu, Katie Rust, Megan L. Noonan, Kenneth E. White, Karl J. Lewis, Uma Sankar, Julia M. Hum, Gregory Jaye Bix, Danielle Wu, Alexander G. Robling, , William R. Thompson

This study identifies a novel mechanosensory complex in osteocytes involving perlecan and α2δ1 subunit, showing that gabapentin disrupts their interaction and impairs bone mechanosensation.

Biomolecules · Dec 12, 2022Peer-reviewedMolecular Biology

An Osteocalcin-deficient mouse strain without endocrine abnormalities

Cassandra R. Diegel, Steven Hann, Ugur M. Ayturk, Jennifer C. W. Hu, Kyung‐Eun Lim, Casey J. Droscha, Zachary Madaj, Gabrielle E. Foxa, Isaac Izaguirre, VAI Vivarium and Transgenics Core, Noorulain Paracha, Bohdan Pidhaynyy, Terry Lynne Dowd, Alexander G. Robling, Matthew L. Warman, Bart O. Williams

This paper describes a new OCN-deficient mouse strain with no endocrine abnormalities, contrasting previous findings, and donates the strain for further study.

PLoS Genetics · May 28, 2020Peer-reviewedGenetics

Skeletal Cell YAP And TAZ Redundantly Promote Bone Development By Regulation Of Collagen I Expression And Organization

Christopher D. Kegelman, Devon E. Mason, James H. Dawahare, Genevieve D. Vigil, Scott S. Howard, Teresita Bellido, Alexander G. Robling, Joel D. Boerckel

This study shows YAP and TAZ redundantly promote bone development by regulating collagen I expression and organization through TEAD4 binding.

bioRxiv · May 30, 2017 · PreprintDevelopmental Biology