Author

Shirley ShiDu Yan

University of KansasORCID 0000-0002-5434-7216Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:imaging· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1 Approach:statistical model· 1

Frequent Co-Authors

Jaichandar SubramanianiD· 2 shared Suraj Niraula· 2 shared Adam S. SmithiD· 1 shared Agnieszka StaniszewskiiD· 1 shared Alexandre A. Sosunov· 1 shared Alice LepelleyiD· 1 shared Andrew F. TeichiD· 1 shared Fang DuiD· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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2 shared papersNeuroscience

Jaichandar SubramanianORCID

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3 papers

Recent·Most cited

NeuroscienceJournal of Alzheimer s DiseasePeer-reviewedAug 7, 2024

Functional connectivity favors aberrant visual network c-Fos expression accompanied by cortical synapse loss in a mouse model of Alzheimer's disease.

Oliver J. L’Esperance, J. McGhee, Garett Davidson…·Jaichandar Subramanian

The paper investigates c-Fos expression and synaptic changes in a mouse Alzheimer's model, finding that functional connectivity predicts c-Fos abnormalities and synaptic weakening in the visual cortex.

9 citations

mouseimaging

NeuroscienceJournal of NeurosciencePeer-reviewedNov 27, 2023

Amyloid pathology impairs experience-dependent inhibitory synaptic plasticity

Suraj Niraula, Shirley ShiDu Yan, Jaichandar Subramanian

This study shows that amyloid pathology impairs inhibitory synaptic plasticity in response to visual deprivation, disrupting synapse adaptation without affecting baseline dynamics.

6 citations

mouseimaging

NeurosciencebioRxivPreprintJan 9, 2020

ECSIT prevents Alzheimer's disease pathology by regulating neuronal mitochondrial ROS and mitophagy

Alice Lepelley, Lauren S. Vaughn, Agnieszka Staniszewski…·Sankar Ghosh

This study shows that ECSIT regulates neuronal mitochondrial ROS and mitophagy, and its reduction contributes to Alzheimer's disease pathology.

3 citations

human