Alica M. Goldman

A population code for semantics in human hippocampus

The paper investigates how semantic information is encoded in human hippocampal activity during speech processing, finding that distributed neural patterns capture semantic relationships similar to language models.

Pathogenic Epilepsy Gene Variant Prevalence and Penetrance Among U.S. Military Veterans in the Million Veteran Program Cohort

The paper examines the prevalence and penetrance of pathogenic variants in epilepsy genes among U.S. military Veterans, finding increased epilepsy risk for carriers of certain variants.

Electrophysiological features of signals recorded from white matter

The paper analyzes electrophysiological signals from white matter and finds they have distinct spectral features and higher complexity compared to gray matter.

Shared neural geometries for bilingual semantic representations in human hippocampal neurons

The paper investigates how bilinguals represent semantic concepts in two languages, finding shared neural geometries that support translation despite language-specific neuron responses.

A semantotopic map in human hippocampus

The paper maps semantic organization in the human hippocampus, finding semantotopic patterns and multiscale semantic similarity along the mediolateral axis.

Linguistic contextualization in the human hippocampus

The paper shows that hippocampal neurons encode word position and context in a way similar to self-attention in large language models.

Plasticity and Language in the Anesthetized Human Hippocampus

This study shows that the human hippocampus can process oddball tones and language stimuli, including semantic and grammatical features, even under general anesthesia.

Exome sequencing of 20,979 individuals with epilepsy reveals shared and distinct ultra-rare genetic risk across disorder subtypes

This study identifies shared and distinct ultra-rare genetic risk factors for epilepsy subtypes using whole-exome sequencing of 20,979 patients and 33,444 controls.

Highly clustered de novo frameshift variants in the neuronal splicing factor NOVA2 result in a specific abnormal C terminal part and cause a severe form of intellectual disability with autistic features.

The paper identifies de novo frameshift variants in NOVA2 causing a severe neurodevelopmental disorder with intellectual disability and autistic features, and shows altered RNA splicing and functional deficits.