Author

Diane M. Armao

University of North Carolina at Chapel HillORCID 0000-0002-5540-5422Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:protein folding & dynamics· 2 Assay:proteomics· 2 Research Focus:cell-cell communication· 1 Research Focus:chromatin & epigenetic regulation· 1

Frequent Co-Authors

Cassandra L. PhillipsiD· 2 shared Natasha Tasheva SnideriD· 2 shared Baggio EvangelistaiD· 1 shared C.C. So· 1 shared Chih-Hsuan HsuiD· 1 shared Jonathan Harrison· 1 shared Maryam FaridounniaiD· 1 shared Meredith F. GillisiD· 1 shared

Activity by Year

2024-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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2 shared papersCell Biology

Cassandra L. PhillipsORCID

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2 papers

Recent·Most cited

Cell BiologyEuropean Journal of Cell BiologyPeer-reviewedJul 16, 2025

The Kelch 3 motif on gigaxonin mediates the interaction with NUDCD3 and regulates vimentin filament morphology

Cassandra L. Phillips, C.C. So, Meredith F. Gillis…·Natasha Tasheva Snider

This study identifies the Kelch 3 motif on gigaxonin as critical for its interaction with NUDCD3 and regulation of vimentin filament morphology.

1 citation

humanproteomics

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Cell BiologybioRxivPreprintSep 3, 2024

Gigaxonin, mutated in Giant Axonal Neuropathy, interacts with TDP-43 and other RNA binding proteins

Cassandra L. Phillips, Maryam Faridounnia, Rachel A. Battaglia…·Natasha Tasheva Snider

This study identifies RNA-binding protein interactions of gigaxonin, a protein mutated in Giant Axonal Neuropathy, revealing potential mechanisms linking its dysfunction to neurodegeneration.

DOI 10.1101/2024.09.03.611033

humanproteomics

𝕏 PostBluesky