Author

Peter A. Kanetsky

Moffitt Cancer CenterORCID 0000-0002-5567-9618Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Genetics· 2 Genetic & Genomic Medicine· 1

Signature Assays, Organisms & Methods

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Assay:GWAS summary statistics· 3 Organism:human· 3 Approach:statistical model· 3 Research Focus:GWAS / association mapping· 2 Research Focus:polygenic risk· 2 Research Focus:fine-mapping· 1 Approach:machine learning (classical)· 1

Frequent Co-Authors

David Timothy BishopiD· 3 shared Christopher Ian AmosiD· 2 shared David C. WhitemaniD· 2 shared David L. DuffyiD· 2 shared Dirk SchadendorfiD· 2 shared Douglas F. EastoniD· 2 shared Florence DémenaisiD· 2 shared Graham J. ManniD· 2 shared

Activity by Year

2018-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersGenetics

David Timothy BishopORCID

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3 papers

Recent·Most cited

Genetic & Genomic MedicinemedRxivPreprintOct 15, 2026

Melanoma germline genetics: immune, cancer driver, and Wnt signaling genes

G J M Shanika R Jayasinghe, Jianxin Shi, Rohit Thakur…·Nhan Do

This paper identifies 116 risk loci for melanoma through GWAS, links susceptibility to immunity and Wnt signaling, and develops a PRS improving risk prediction.

DOI 10.64898/2026.10.14.26365068

humanGWAS summary statistics

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GeneticsNature CommunicationsPeer-reviewedJul 3, 2020

Assessment of Polygenic Architecture and Risk Prediction based on Common Variants Across Fourteen Cancers

Yan Zhang, Amber N. Hurson, Haoyu Zhang…·Colon Cancer Family Registry (CCFR)

This study assesses polygenicity and risk prediction using common variants across 14 cancers, finding varying numbers of susceptibility variants and risk score effectiveness.

137 citationstop 1%

humanGWAS summary statistics

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GeneticsNature CommunicationsPeer-reviewedNov 8, 2018

Novel pleiotropic risk loci for melanoma and nevus density implicate multiple biological pathways

David L. Duffy, Gu Zhu, Xin Li…·Nicholas Orr

This paper identifies novel pleiotropic risk loci for melanoma and nevus density, implicating multiple biological pathways and gene expression under MITF regulation.

117 citationstop 2%

humanGWAS summary statistics

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