Author

Tao Wang

Sir Run Run Shaw HospitalORCID 0000-0002-6348-2794Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Tao Wang16 papers · Baylor College of Medicine

Biology Disciplines

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Immunology· 1 Molecular Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

Aliya IzumiiD· 1 shared Anthony O GramoliniiD· 1 shared Boris Hinz· 1 shared Canjun ZhuiD· 1 shared Cha Zhang· 1 shared Chia‐Wei ChangiD· 1 shared Cong YiniD· 1 shared Cristine J ReitziD· 1 shared

Activity by Year

2020-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 13 mutual co-author links

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1 shared paperPhysiology

Aliya IzumiORCID

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3 papers

Recent·Most cited

Latent Transforming Growth Factor Binding Protein-2 promotes microfibrillar TGF-β bioavailability to drive pathologic fibrosis in remote regions of infarcted hearts

Fahad Ehsan, Michelle Di Paola, Micael Dewar, Hangjun Zhang, Uros Kuzmanov, Yu-Qing Zhou, Cristine J Reitz, Haisam Shah, Tao Wang, Jingzhi Liu, Aliya Izumi, Rundong Hu, Joanna Hendy, Fareshteh Sadat Younesi, Mark K. Friedberg, , Scott P Heximer

This study shows that LTBP2 promotes fibrosis in remote myocardium after MI by sequestering latent TGF-β, and its deletion reduces fibrosis and improves cardiac function.

bioRxiv · Nov 3, 2026 · PreprintPhysiology

Type-I interferon priming signal balances anti-bacterial and anti-tumor trained immunity in alveolar macrophages

Tao Wang, Jinjing Zhang, Yuxuan Miao, Yanling Wang, Ying Li, Lu Wang, Yuanyuan Liu, Chia‐Wei Chang, Enguo Chen, Yushi Yao

This study shows that type-I interferon signaling from influenza infection primes alveolar macrophages to develop balanced anti-bacterial and anti-tumor trained immunity.

bioRxiv · Jun 14, 2025 · PreprintImmunology

α-ketoglutaric acid stimulates muscle hypertrophy and fat loss through OXGR1-dependent adrenal activation

Yexian Yuan, Pingwen Xu, Qingyan Jiang, Xingcai Cai, Tao Wang, Wentong Peng, Jiajie Sun, Canjun Zhu, Cha Zhang, Dong Yue, Zhi‐Hui He, Jinping Yang, Yuxian Zeng, Man K. Du, Fenglin Zhang, , Gang Shu

This study identifies α-ketoglutaric acid (AKG) as a metabolite that promotes muscle hypertrophy and fat loss via OXGR1-dependent adrenal activation in response to resistance exercise.

The EMBO Journal · Feb 27, 2020Peer-reviewedMolecular Biology