Author

Mi Tian

St. Jude Children's Research HospitalORCID 0000-0002-6446-1995Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Approach:experimental assay· 1 Organism:mouse· 1 Approach:statistical model· 1

Frequent Co-Authors

Ben YoungbloodiD· 1 shared Danielle SeilheaniD· 1 shared David KlatzmanniD· 1 shared Gwendolyn Marguerit· 1 shared Gwladys FourcadeiD· 1 shared Laura MoutoniD· 1 shared Michèlle RosenzwajgiD· 1 shared Nicolas CoatnoaniD· 1 shared

Activity by Year

2025-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperImmunology

Ben YoungbloodORCID

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1 paper

Recent·Most cited

Maternal immune activation imprints a regulatory T cell deficiency in offspring that drives an autism-like phenotype

Pierre Ellul, Gwladys Fourcade, Vanessa Mhanna, Nicolas Coatnoan, Susana Bodula, Danielle Seilhean, Laura Mouton, Gwendolyn Marguerit, Richard Delorme, Mi Tian, Ben Youngblood, Michèlle Rosenzwajg, Nicolas Tchitchek, David Klatzmann

Maternal immune activation causes Treg deficiency in offspring, leading to brain inflammation and autism-like behaviors, which can be reversed by Treg stimulation.

bioRxiv · Jan 6, 2025 · PreprintImmunology