Author

Toshiki Kameyama

Fujita Health UniversityORCID 0000-0002-7348-7572Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Molecular Biology· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:multiome· 1 Approach:statistical model· 1

Frequent Co-Authors

Akila MayedaiD· 1 shared Akira NakashimaiD· 1 shared Hiroshi NagasakiiD· 1 shared Hisateru YamaguchiiD· 1 shared Kanako SaitoiD· 1 shared Ken‐ichi FujitaiD· 1 shared Miho KawataiD· 1 shared Yu Kodani· 1 shared

Activity by Year

2021-2025 · peak 1/yr

Co-Author Collaboration Network

9 top collaborators · 18 mutual co-author links

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1 shared paperMolecular Biology

Akila MayedaORCID

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2 papers

Recent·Most cited

NT5DC2 downregulation suppresses monoamine oxidase activity and increases catecholamine levels in PC12D cells

Hisateru Yamaguchi, Miho Kawata, Yu Kodani, Kanako Saito, Toshiki Kameyama, Hiroshi Nagasaki, Akira Nakashima

This study shows that NT5DC2 downregulation reduces MAO A activity and increases catecholamine levels in PC12D cells.

Molecular Biology Reports · Aug 2, 2025Peer-reviewedNeuroscience

The Exon Junction Complex Core Represses Caner-specific Mature mRNA Re-splicing: A Potential Key Role in Terminating Splicing

Yuta Otani, Ken‐ichi Fujita, Toshiki Kameyama, Akila Mayeda

The paper identifies the exon junction complex core as a repressor of cancer-specific mRNA re-splicing, showing that its knockdown mimics cancer-like splicing in normal cells.

International Journal of Molecular Sciences · Jun 17, 2021Peer-reviewedMolecular Biology