Author

Ken‐ichi Fujita

Fujita Health UniversityORCID 0000-0002-3104-5274Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Molecular Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:multiome· 2 Research Focus:cell-cell communication· 1 Research Focus:gene regulatory network inference· 1 Research Focus:spatial domain detection· 1 Approach:statistical model· 1

Frequent Co-Authors

Akila MayedaiD· 2 shared Fumito Hori· 1 shared Hanae Yoshioka· 1 shared Hideo MatsudaiD· 1 shared Kotaro Harada· 1 shared Moe Yokoshi· 1 shared Naoko FujiwaraiD· 1 shared Seiji MasudaiD· 1 shared

Activity by Year

2021-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersMolecular Biology

Akila MayedaORCID

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2 papers

Recent·Most cited

Two closely related RNA helicases, UAP56 and URH49, couple mRNA splicing and export pathways with their distinct complexes

Ken‐ichi Fujita, Kotaro Harada, Yuya Ikeda, Hanae Yoshioka, Moe Yokoshi, Fumito Hori, Naoko Fujiwara, Shigeto Seno, Hideo Matsuda, Yukio Kawahara, Akila Mayeda, Seiji Masuda

This study identifies three splicing regulation patterns mediated by UAP56 and URH49 and their association with mRNA export pathways.

bioRxiv · Oct 29, 2026 · PreprintMolecular Biology

The Exon Junction Complex Core Represses Caner-specific Mature mRNA Re-splicing: A Potential Key Role in Terminating Splicing

Yuta Otani, Ken‐ichi Fujita, Toshiki Kameyama, Akila Mayeda

The paper identifies the exon junction complex core as a repressor of cancer-specific mRNA re-splicing, showing that its knockdown mimics cancer-like splicing in normal cells.

International Journal of Molecular Sciences · Jun 17, 2021Peer-reviewedMolecular Biology